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PharmD Signal™ · Quick Card

⚖️ Corticosteroid Equivalencies

Reviewed by a PharmD, BCCCP · Updated Jul 2026
Glucocorticoid Equivalency Table
Short-acting (8-12 h) Intermediate-acting (12-36 h) Long-acting (36-72 h)
DrugEquiv. Dose (mg)Relative GC PotencyRelative MC PotencyDurationBiologic t½
Hydrocortisone2011Short8-12 h
Cortisone250.80.8Short8-12 h
Prednisone540.8Intermediate12-36 h
Prednisolone540.8Intermediate12-36 h
Methylprednisolone450.5Intermediate12-36 h
Triamcinolone450Intermediate12-36 h
Dexamethasone0.7525-300Long36-72 h
Betamethasone0.625-300Long36-72 h

GC = glucocorticoid; MC = mineralocorticoid. Equivalencies apply to anti-inflammatory / immunosuppressive effects only and are approximate. They do NOT apply linearly at very high (“pulse”) doses. MC potency values vary by source (e.g., prednisone MC is cited as 0.3-0.8 depending on assay and reference); clinically, MC effects are significant for hydrocortisone/cortisone, moderate for prednisone/prednisolone, and negligible for methylprednisolone through dexamethasone. Fludrocortisone is omitted (used exclusively for MC replacement; GC ~10, MC ~125).

Conversion Method
StepActionExample
1Identify the current drug and daily dosePrednisone 40 mg/day
2Convert to hydrocortisone equivalents
current dose × (20 ÷ current equiv. dose)
40 × (20 ÷ 5) = 160 mg hydrocortisone equiv.
3Convert to the target drug
HC equiv. × (target equiv. dose ÷ 20)
160 × (0.75 ÷ 20) = 6 mg dexamethasone

Or use the shortcut: current dose × (target equiv. dose ÷ current equiv. dose). Example: 40 mg prednisone × (0.75 ÷ 5) = 6 mg dexamethasone.

Common Conversions at a Glance
FromToMultiply by
PrednisoneMethylprednisolone× 0.8 (5 mg pred = 4 mg methylpred)
PrednisoneDexamethasone× 0.15 (5 mg pred = 0.75 mg dex)
PrednisoneHydrocortisone× 4 (5 mg pred = 20 mg HC)
MethylprednisolonePrednisone× 1.25 (4 mg methylpred = 5 mg pred)
DexamethasonePrednisone× 6.67 (0.75 mg dex = 5 mg pred)
HydrocortisonePrednisone× 0.25 (20 mg HC = 5 mg pred)
Physiologic vs. Pharmacologic Dosing
CategoryHydrocortisone EquivalentPrednisone EquivalentClinical Context
Physiologic replacement15-25 mg/day3.75-6.25 mg/dayAdrenal insufficiency (primary or secondary)
Stress dose (minor procedure)50 mg IV ×1Minor surgery, dental procedures in AI patients
Stress dose (major surgery / critical illness)50 mg IV q8h (or 100 mg q8h)Major surgery, septic shock (SSC: 200 mg/day IV)
Low-dose anti-inflammatory5-10 mg/dayRA maintenance, PMR
Moderate immunosuppressive20-40 mg/dayAcute exacerbations (COPD, asthma), ITP
High-dose immunosuppressive1-2 mg/kg/daySLE nephritis, vasculitis, transplant rejection
Pulse therapyMethylpred 500-1000 mg IV daily ×3-5 dMS relapses, severe lupus, optic neuritis. Equivalency table does NOT apply at pulse doses.
Taper Considerations
QuestionGuidance
When is a taper needed?Generally required if steroids used >3 weeks at any dose, or >1 week at ≥prednisone 20 mg/day. Short bursts (≤7-10 days, ≤40 mg/day prednisone) can usually be stopped abruptly.
Why taper?Exogenous steroids suppress the HPA axis. Abrupt withdrawal after prolonged use risks adrenal crisis (hypotension, hypoglycemia, shock).
General approachReduce by 10-20% every 1-2 weeks while monitoring for symptoms of adrenal insufficiency (fatigue, nausea, hypotension, arthralgias) and disease flare.
Below physiologic rangeOnce prednisone reaches ~5-7.5 mg/day (physiologic equivalent), slow the taper further (1 mg decrements q2-4 wk) and consider AM cortisol or cosyntropin stimulation test to assess HPA recovery before discontinuing.
Long-acting agentsDexamethasone causes more HPA suppression per equivalent dose due to longer biologic half-life. Consider switching to shorter-acting agent (prednisone/prednisolone) before tapering.
Key Clinical Pearls
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Prednisone is a prodrug: it must be hepatically converted to prednisolone (the active form). In severe liver dysfunction (cirrhosis, acute liver failure), use prednisolone or methylprednisolone instead.
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Mineralocorticoid effects matter: hydrocortisone and cortisone have significant MC activity (Na+ retention, K+ wasting, edema, hypertension). At high doses, consider switching to methylprednisolone or dexamethasone (near-zero MC effect) to avoid fluid overload.
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Dexamethasone ≠ prednisone for all indications: the equivalency table gives anti-inflammatory equivalence, but clinical trial dosing for specific conditions (e.g., dex 6 mg in RECOVERY for COVID, dex 40 mg x4 d for myeloma) is NOT derived from simple conversion. Use the protocol-specific dose.
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Steroid-induced hyperglycemia: most pronounced with intermediate-acting agents (prednisone, methylprednisolone) given as a morning dose, producing afternoon/evening BG spikes. NPH insulin dosed in the AM mirrors this pattern and is a first-line choice. Check BG even in non-diabetic patients on ≥prednisone 20 mg/day.
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Stress dosing for chronic steroid patients: any patient on ≥prednisone 5 mg/day (or equivalent) for 3+ weeks should be assumed to have HPA suppression and needs stress-dose steroids for surgery, trauma, or critical illness. Do not rely on a “normal” random cortisol.
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IV-to-PO conversion: methylprednisolone IV and PO are 1:1 (bioavailability >80%). Hydrocortisone IV to PO is also 1:1. Dexamethasone IV to PO is 1:1 (near 100% oral bioavailability). Predniso(lo)ne IV formulations are less common in the US; oral bioavailability is excellent.
Educational quick reference for licensed clinicians. Not a substitute for prescribing information, clinical judgment, or institutional protocols. Verify against current labeling and patient-specific factors. © 2026 PharmD Signal.
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